Title: The Lack of Love and Iron, The two causes of Alzheimer’s

Abstract:

Objective: By the lack of initiative by force (Faith) Iron man lives. Iron deficiency causes anemia, anemia causes dementia, Alzheimer's dementia and Alzheimer’s produces cognitive impairment in memory produces bases. Well, hear him. The Iron Will Alkaline, the answer is yes.
Methodology: On the basis of Love and the use of Iron and its allies, which are the B vitamins, Vitamin C, E, and vitamin A. It is necessary to remember that there is to try to fight the greatest sustenance Anemia in all its contrarestantes.
Conclusion: The theory focuses on the oxygenation of the blood, which must be done, where the Warburg Alkaline Diet is demonstrated, among other factors it is necessary to emphasize the oxygenation that consists of the mental and physical, which is reduced in Sleeping correctly, Warburg Alkaline Diet, Drink Enough Water, Make Walks or Moderate Exercises, Comfort and Drink Iron, Vitamin C, Vitamin E, Complex B, and Vitamin A. All this consists of Producing New Oxygen.

Biography:

I Am The Creator Of Global Zionist Nemruthism And Towards Moral Sociology: Historical Measurementism. I have done more than fifteen theories and A cure: ALZ, CANCER, TOE.

Title: Prevalence, Risk factors and Antibiotic Resistance of Staphylococcus aureus and MRSA nasal carriage among healthy population in Ibadan, Nigeria

Abstract:

Background: Nasal carriage of Community-Acquired Methicillin-resistance Staphylococcus aureus (CA-MRSA) is recognized for its rapid community spread and tendency to cause various infections especially in communities with a large population where personal hygiene is poor. We sought to investigate the prevalence and evaluated the possible risk factors of CA-MRSA among the healthy population.

Methods: Nasal swabs collected from 392 males and 308 females using the multi-stage sampling technique were cultured for Staphylococcus aureus. Isolates were identified by conventional biochemical tests, Microbact™ 12S identification kit, and confirmed with 16SrRNA. Antibiotic susceptibility testing was performed using the Kirby-Bauer disc diffusion technique. Finally, isolates were further investigated for methicillin resistance by using the cefoxitin disk diffusion test followed by polymerase chain reaction amplification of Meca and Nuc genes. Proportions were tested using Chi-Square and Fisher’s Exact Probability Test in Epi inform.

Results: The results showed 31.9% and 9.4% prevalence of S. aureus nasal carriage and Methicillin-resistance Staphylococcus aureus respectively. Low educational background (ϰ² =36.817, P ˂ .001), age >40-50 years (ϰ² = 8.849, P = .003), recent antibiotics use (ϰ² = 7.556, P = .006), recent hospital visitation (ϰ² = 8.693, P = .003) and male gender (ϰ² = 9.842, P = .002) are significantly associated with CA-MRSA. The results of this research study show that CA-MRSA are highly multi-drug resistant.

Conclusion: The study established a high prevalence and resistance burden of CA-MRSA in the population; this poses a serious public health concern in the region and necessitates the demands for continuous surveillance on the colonization state of CA-MRSA to restrict the transmission of the bacterium in the community.

Biography:

I'm a driven Doctoral Researcher, a Global Health professional with expertise in Infectious diseases & Antimicrobial Stewardship.
A Global Outreach Contributing Member of American Society for Microbiology (ASM) and a member of the African Society for Laboratory Medicine (ASLM).
A diligent and innovative individual with over 7 years of a broad range of unique experiences both with university-based and clinical researchers. Specifically, my previous role as a research assistant working on an internationally-funded one-health based surveillance and in my current role on AMR enteric pathogens enabled me to hone my laboratory, analytical, and data management skills. I am increasingly developing advanced knowledge and transferable research skills.

• I am also skilled in biological and biochemical assay development, Epidemiology, Molecular biology, and gene expression profiling.
• Possess the cultural competence and the interpersonal skills to function effectively in a multi-cultural and multidisciplinary team.

Specialties: Infectious Diseases, infection control, antimicrobial resistance, antimicrobial stewardship, and public health surveillance.

Title: Ethical Issues regarding embryonic Stem cells Research

Title: Finding New Ways to Improve the Heal of the Physical and Mental Wounds An overview

Biography:

Mahira Amirova, an Assoc. Prof. In Azerbaijan Medical University, 1966. In 1988, graduated Az.Med.University pharmaceutical faculty. In 1988-1993 worked at theme "Atherosclerosis correction by tritherpenoids". PhD in biological science. Nationality: azerbaijani, Is a co-author of 2 books:"Biochemical laboratory classes" and " "Biochemistry laboratory manual and theory and methods", an editor of book "Pathological and clinical biichemistry" An author of teaching aids: "Biichemical mechanism of biotransformation and elimination of xenobiitics" An author of teaching aids: " Biochemical aspects of aging", "Some points in biochemistry and pathobiochemistry of cell", "Role of apoptosis in pathogenesis of certain diseases", "Biochemical aspects of ethanol action and metabolism", An author of annual biochemistry test-books for students of treatment, dental, pharmaceutical faculties, and test-book in clinical biochemistry. An expert in "State Comission for post-graduates"

Title: The Lack of Fam83h Mediated Reduction of Wnt/?-Catenin Signaling Pathway and Expression Levels of Dental Mineralization Genes

Abstract:

Background: FAM83H has been identified as an essential gene for dental enamel formation and may be related to Wnt/β-catenin.
Methods: levels of Fam20a, Dspp, Dmp1, Enam, Ambn, Sppl2a, Mmp20, Fgf10, and the mediators of Wnt/β-catenin pathway were measured in the dental root of both Fam83h-KnockOut and wild-type mice by using Q-PCR at 5,11 and 18 days after birth. The expression of Fgf10 and the mediators of Wnt/β-catenin were also evaluated in the skin of KnockOut and wild-type mice by using Q-PCR and also, The histology of hair follicles was compared. the Fam83h-KnockOut mice recruited in this study confirmed by Sanger sequencing and western blot analysis.
Results: Our results showed that Ambn, Mmp20, Dspp, and Fgf10 significantly reduced in Fam83h-KnockOut mice in the dental root, associated with marked reduction of CK1a, CK1e, and β-catenin expression in Fam83h-KnockOut mice in the dental root. The Fgf10, CK1a, CK1e, and β-catenin were significantly decreased in the skin of Fam83h-KnockOut mice. In the absence of Fam83h, the accumulation of unemployed CK1a is expected to elevate the β-catenin destruction complex. The reduction of CK1e may also decrease the signaling of Dvl-1, leading to the suppression of the Wnt/β-catenin pathway. Simultaneous reduction of both mineralization genes and Wnt/β-catenin pathway due to the absence of Fam83h has a potential to be related to the deficiency of dental formation and mineralization. Further, concurrent reduction of Fgf10 gene expression and Wnt/β-catenin pathway may also affect the maturation of hair follicles as confirmed by histological examination.
Conclusion: it seems that in the lack of Fam83h, dental mineralization is induced by simultaneous decrease of Wnt / β-catenin mediators and the mineralization-related genes, suggesting to act in a cumulative effect manner and probably behave as a multi-factorial trait.

Biography:

I'm Sherko Nasseri, Ph.D. in molecular medicine. Assistant Professor, from Kurdistan University of Medical Sciences, Iran. During my Ph.D. Thesis, I worked on the generation of Fam83h Knockout Mice by using the CRISPR/Cas9 method. The absence of Fam83h these mice has the scruffy cover, dry eye phenotypes, and also these mice were smaller than the same age normal mice. In continuation of this project, we found that the WNT/B-Catenin pathway decreased. The Fam83h gene has a high expression level in the gastrointestinal tract. Given all that has been said, it brings me a critical question that how the fam83h gene play role in the gastrointestinal tract?